IMBABY, I., NEGM, S., HERMAS, G., RAGHEB, D. (2007). PATHOGENIC SPECIALIZATION AND PATHOTYPE DISTRIBUTION OF PUCINIA GRAMINIS TRITICIIN EGYPT IN 2005/2006 AND POSTULATED GENES OF RESISTANCE IN SOME WHEAT GENOTYPES. test, 85(4), 1169-1184. doi: 10.21608/ejar.2007.227572
IBRAHIM A. IMBABY; SOBHY S. NEGM; GAMALAT HERMAS; DOAA RAGHEB. "PATHOGENIC SPECIALIZATION AND PATHOTYPE DISTRIBUTION OF PUCINIA GRAMINIS TRITICIIN EGYPT IN 2005/2006 AND POSTULATED GENES OF RESISTANCE IN SOME WHEAT GENOTYPES". test, 85, 4, 2007, 1169-1184. doi: 10.21608/ejar.2007.227572
IMBABY, I., NEGM, S., HERMAS, G., RAGHEB, D. (2007). 'PATHOGENIC SPECIALIZATION AND PATHOTYPE DISTRIBUTION OF PUCINIA GRAMINIS TRITICIIN EGYPT IN 2005/2006 AND POSTULATED GENES OF RESISTANCE IN SOME WHEAT GENOTYPES', test, 85(4), pp. 1169-1184. doi: 10.21608/ejar.2007.227572
IMBABY, I., NEGM, S., HERMAS, G., RAGHEB, D. PATHOGENIC SPECIALIZATION AND PATHOTYPE DISTRIBUTION OF PUCINIA GRAMINIS TRITICIIN EGYPT IN 2005/2006 AND POSTULATED GENES OF RESISTANCE IN SOME WHEAT GENOTYPES. test, 2007; 85(4): 1169-1184. doi: 10.21608/ejar.2007.227572

PATHOGENIC SPECIALIZATION AND PATHOTYPE DISTRIBUTION OF PUCINIA GRAMINIS TRITICIIN EGYPT IN 2005/2006 AND POSTULATED GENES OF RESISTANCE IN SOME WHEAT GENOTYPES

Egyptian Journal of Agricultural Research
Article 4, Volume 85, Issue 4, December 2007, Pages 1169-1184    PDF (5.27 M)
Document Type: Original Article
DOI: 10.21608/ejar.2007.227572
Authors
IBRAHIM A. IMBABY; SOBHY S. NEGM; GAMALAT HERMAS; DOAA RAGHEB
Wheat Diseases Research Section, Plant Pathology Research Institute, Agricultural Research Center, Giza, Egypt
Abstract

Wheat stern rust surveys gave evidence to the presence of nine and sixteen physiologic races in 2005 and 2006 growing season, respectively. In the first season, race 34 was the predominant (S1.980/0) followed by race 11 (25.25%). Two-hundred and two virulence phenotypes were detected among these races. The most effective resistance genes were Sr 26, 29, 11, 3.1, 35, and 9e. In the second season, race 15 was the predominant (27.26%) followed by race 11 (24.230/0). Sixty-six virulence phenotypes were detected under these races. 5r26 was the most effective gene. Most of these genes were postulated to be present, either alone or in combinations, in the genotypes tested.
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